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D-BOL
Compound: Methandrostenolone
Dianabol Guide
The Anabolic Catalyst
Dianabol is the legendary "Breakfast of Champions," specifically engineered to enhance nitrogen retention and facilitate massive glycogen storage. As a 17-alpha-alkylated oral compound, it bypasses liver filtration to deliver rapid, supraphysiological strength gains.

Anabolic Rating

90-210

Highly Potent

Androgenic Rating

40-60

Reduced Impact

Chemical Formula

C20H28O2

Methylated

Half-Life

3-5 Hours

Rapid Clearance

System.Core.Steroid_Database
Information Hub

Anabolic Baseline

100pts

Androgenic Ref

100pts

📊Overview
🧬Testosterone💊Dianabol
🔥Trenbolone
⚡Anavar
Database Sync: Optimized
Security Protocol: WADA Standard
C20-H28-O2
What is Dianabol?
Methandrostenolone (Dbol) is an orally active, C17-alpha alkylated anabolic steroid. Originally developed to provide a competitive edge in athletics, it is renowned for its ability to induce rapid protein synthesis and massive glycogen retention.
MASS
ANB
Primary Effects

  • • Dramatic Nitrogen Retention

    Keeps muscle tissue in a constant anabolic state.

  • • Enhanced Glycogenolysis

    Dramatically increases strength and muscle fullness.

  • • Rapid Strength Spikes

    Noticeable power increases within 48-72 hours.

  • • Protein Synthesis Acceleration

    Fast-tracks recovery of damaged muscle fibers.

ORAL
LOG
Administration

  • • 17-aa Methylation

    Orally active; survives first-pass liver metabolism.

  • • Pulse-Dosing Required

    Short half-life (3-6h) requires split daily doses.

  • • Kickstart Protocol

    Commonly used in first 4-6 weeks of an injectable cycle.

  • • Bioavailability

    High absorption rate when taken on an empty stomach.

Impact & Hypertrophy
POW
⚡
Strength Logic
  • Force ProductionULTRA-FAST
  • Intracellular Hydration+ 40%
  • Glycogen SaturationOPTIMIZED
  • Lifting ThresholdINCREASED
MET
🧬
Anabolic Sync
  • Nitrogen BalancePositive+
  • Protein SynthesisCRITICAL
  • Cortisol SuppressionACTIVE
  • IGF-1 ProductionELEVATED
REC
🔥
System Vitality
  • Joint LubricationFLUID+
  • Neural DriveAGGRESSIVE
  • Dopamine BaselinePEAK
  • Recovery Window- 50% TIME
Toxicity // PathologyCritical System Warning: Methandrostenolone
LIVER STRAIN: SEVERE
Acute Response
01. Common Disruptions

Hepatic Load

Elevated ALT/AST Enzymes

Renal System

Water Retention & Lower Back 'Pumps'

Cardiovascular

Rapid Blood Pressure Spikes

Gastrointestinal

Loss of Appetite / Nausea

High Risk
02. Critical Failure

Estrogenics

17α-Methylestradiol (Strong Gyno Risk)

Lipid Profile

Aggressive HDL Suppression

Endocrine

Instant HPTA Shutdown

Epidermal

Cystic Acne & Acceleration of MPB

Pharmacological Audit: D-BOL
METHANDRO STENOLONE
Compounding StatusGrade-A Toxicant
☢️
False Intelligence
"Orals are safer because there are no needles."
Biological RealityDianabol is **C17-Alpha Alkylated**. To survive digestion, it is chemically armored, forcing the liver to process it multiple times. This creates **intrahepatic cholestasis**, causing more internal damage than many injectable oils.
Common Fallacy
"The 10lbs I gained in 2 weeks is pure muscle."
Biological RealityDianabol causes massive **Intracellular Water Retention**. Most "weight" gained is glycogen and fluid stored in the sarcoplasm. Once the cycle ends and estrogen drops, up to **60-70% of that weight vanishes** instantly.
DB
Tactical Mitigation StackCompound: Methandrostenolone (Dianabol)
01. Hepatic Protection
Mandatory

TUDCA (750mg)

Critical for bile flow; prevents cholestasis induced by 17aa alkylation.

Essential

NAC (1200mg)

Precursor to Glutathione; primary defense against oxidative liver stress.

Critical

Arimidex (0.5mg EOD)

Blocks potent Methylestradiol conversion (Gyno prevention).

Optional

Telmisartan

BP management for the heavy water-retention spikes (The D-Bol Pump).

02. Biological Load

Plasma Half-Life

3-5 Hours

Requires split dosing for stable serum levels.

Anabolic Ratio

90-210

Significantly more potent than standard Test.

Estrogen Potency

Severe

Converts to highly active 17α-methylestradiol.

HPTA Impact

100% Shutdown

Complete suppression within 7 days of use.

!!
Critical Mechanism: Sarcoplasmic Expansion
Dianabol operates via massive nitrogen retention and a drastic increase in glycogen synthesis. While lean tissue is built, the primary "visual" gain is **Sarcoplasmic Hypertrophy** (fluid inside the muscle). Failure to maintain a **High Potassium/Low Sodium** ratio during this protocol will result in severe hypertension and left ventricular strain.
6-8 WeeksMax Cycle Duration
TUDCA/NACNon-Negotiable
Pharma-Grade Analysis
Dianabol // D-BOLIUPAC: 17α-Methyl-17β-hydroxyandrosta-1,4-dien-3-one
Pharmacodynamic Profile
Anabolic Ratiovs. 100 (Testosterone)
210
Androgenic RatioReduced via 1-2 Double Bond
60
17α-AlkylationC17-Alpha-Methyl Group
Present
Half-Life (Oral)Rapid Plasma Clearance
4.5 - 6 Hours
Metabolic Clearance Matrix

Estrogen

High

17α-methylestradiol

Water Ret.

Severe

Intracellular edema

Liver Strain

Critical

AST/ALT Elevation

Blood Pressure

Spiked

Sodium Retention

Dosing Warning

Due to the short half-life, total daily dosage should be split (e.g., 8am / 2pm / 8pm) to avoid serum level crashes and minimize gastric distress.

Hepatic Load

ALT/AST Heavy

Toxicity

Lipid Impact

HDL Suppression

Critical

Conversion

Aromatase Type 1

Biological

SHBG

Heavy Reduction

Mechanism

LEGAL NOTICE:This data is for structural analysis only. Methandrostenolone is a Schedule III substance. Unsupervised use is strictly high-risk.
⚖️
Compliance & Legal Risk
Statutory Framework: Methandrostenolone
Global Criminal Classifications
United States (DEA/FDA)SCHEDULE III
Classified under the **Controlled Substances Act**. Since the FDA withdrew approval for human use in the 1980s, possession is almost exclusively viewed as **Illicit**. Trafficking yields up to 10 years federal imprisonment.
European Union (EMA)RESTRICTED
Varies by member state; however, Germany and France maintain strict **Anti-Doping Laws** where even possession of small quantities can lead to criminal prosecution under the Anti-Doping Act (AntiDopG).
Athletic Sanctions Matrix
!
WADA Prohibited ListCategory S1.1A (Exogenous AAS)
Dianabol metabolites (Epimetendiol) are detectable for up to **19 days** via high-sensitivity Mass Spectrometry. Use triggers an automatic **4-year ban** for first-time offenders under the World Anti-Doping Code.
Detection TechGC/C/IRMS Analysis
Customs FocusHigh Priority (Oral)
Critical Legal Disclosure
As Dianabol is no longer manufactured by major Western pharmaceutical companies, any product currently available is likely of Underground Lab (UGL) origin. This introduces secondary legal risks associated with **organized crime statutes** and extreme health risks including heavy metal poisoning, unlisted ingredients, or septic contamination.
REF: UNODC GLOBAL REPORT ON AASCAS NO: 72-63-9
ORAL_PHASE
Advanced Protocol
Dosage & Administration

Dosing Unit

mg / Day

Route

Oral/C17aa

🧪
Therapeutic & Bridge Range

Clinical Osteoporosis

Single AM Bolus

5-10mg

Historical medical application

The 'Kickstart' Phase

Split 2x Daily

20-30mg

First 4 weeks of cycle

Plateau Breaking

Pre-Workout Only

25mg

CNS stim & glycogen focus

⚡
Hypertrophy Tiers

Mass Accrual

Duration: Max 6 Weeks

30-50mg

High-Level Build

Duration: Liver Protection Required

50-80mg

Extreme Hazard

Duration: Liver Failure Warning

100mg+
⚠️
Cycle length must not exceed 6-8 weeks due to hepatic strain and lipid degradation.
Half-Life Strategy

Dianabol has a 4-6 hour half-life. To maintain Peak Serum Concentration, split the daily dose into 3-4 administrations.

Methylation Impact

The 17-alpha-alkylation allows survival through the first liver pass, but creates significant Hepatocyte Strain.

Estrogen Spike

Dianabol aromatizes into 17α-methylestradiol, which is far more potent than regular estradiol. An AI is usually mandatory.

!
Liver Support Integration RequiredTUDCA 500mg / NAC 1200mg Daily Recommended
System Status: Critical Recovery
HPTA RestorationPost-Methandrostenolone Protocol
The "Dbol Crash" AnalysisDianabol causes rapid shutdown of the Hypothalamic-Pituitary-Testicular Axis. Because its half-life is so short, hormone levels plummet within 24 hours of the last dose, triggering an immediate catabolic spike in **Cortisol**.

24h

Clearance

High

Estrogen Rebound

Ancillary Stack & SERMs
Nolvadex (Tamoxifen)Core SERM
40/40/20/20
Essential for managing the Dbol-induced estrogen rebound and stimulating LH/FSH.
Clomid (Clomiphene)Pituitary Kick
50/50/25/25
Aggressively forces the release of GnRH to restart natural testosterone production.
Cystone / Liver RefreshHepatic Recovery
Daily
Required to clear liver enzymes (ALT/AST) elevated by Dbol's C17-aa nature.
Aromasin (Exemestane)Suicide AI
12.5mg EOD
Used briefly during the transition to prevent 'Moon Face' and gyno during the crash.
Day 1-2 Post Cycle
The TransitionDbol clears the blood rapidly. This is the 'Danger Zone' where muscle flatness and mood drops occur.
Day 3 - Week 3
Aggressive RestartBegin SERM protocol immediately. Focus on high-intensity, low-volume training to maintain gains.
Week 4 - 6
NormalizationTaper dosages. Focus on natural test-boosting micronutrients (Zinc, Magnesium, Vitamin D3).
Week 8
The AuditComprehensive blood panel. If LH/FSH are not in range, a secondary recovery phase is required.
🩸Mandatory Bio-Marker Audit
Do not guess. Dbol is notorious for skewing **Lipid Profiles (HDL/LDL)** and suppressing **Natural Testosterone** more harshly than many injectables. Bloodwork must be performed 4 weeks post-PCT to ensure the HPTA is truly functional.
Liver EnzymesLipid PanelTotal/Free TLH & FSH
HEPATIC-CARDIO DEFENSE SYSTEM
DBOL VIGILANCE
System StatusMonitoring Toxicity
Bio-AlertsActive Scan
Critical Diagnostic Grid
Liver Enzymes (ALT/AST/GGT)
High Risk
Dbol is C17-aa. Monitor GGT specifically—it is a more sensitive marker for cholestatic liver injury.
HEPATOTOXICITY
Lipid Architecture
LDL ↑ HDL ↓
Rapid HDL reduction. Dbol bypasses the liver's lipid regulation, spiking LDL and hardening arteries.
ATHEROGENIC
Kidney Strain (eGFR/BUN)
Filtration
Increased blood pressure from water retention stresses the nephrons. Keep eGFR above 90.
RENAL LOAD
17α-MeE2 (Estrogen)
Aggressive
Potent methyl-estradiol conversion. Causes high water weight, moon-face, and hypertension.
AROMATIZATION
Cycle Log
00
Pre-LoadLiver & Lipid baseline.
04
Toxic PeakLiver enzymes check mandatory.
06
Off-CycleDbol stops; begin PCT.
12
Full AuditVerify cardiovascular recovery.
BP
Critical AlertBlood Pressure & Hypervolemia
Dianabol causes rapid sodium and water retention via its conversion to **17$\alpha$-methylestradiol**. This increases total blood volume, forcing the heart to pump harder. Chronic use without monitoring can lead to **Left Ventricular Hypertrophy (LVH)**. Maintain blood pressure below **130/80 mmHg** using telmisartan or lifestyle modification.
Ref: GGT-PathologyRef: LVH-Cardiac-Scan
Biomarker Integrity: 100% VerifiedLast Protocol Update: Dec 2025
DBOL
POST-ANALYSIS VERDICT
Classification: Hepatotoxic Oral Agent
Extreme Risk Profile
The C17-α Alkylation Penalty
Dianabol is structurally modified to survive the **First-Pass Metabolism**. While this allows for oral bioavailability, it forces the liver to process a compound it cannot easily break down. This creates a state of **Oxidative Stress** that can lead to Peliosis Hepatis or Cholestatic Jaundice if used beyond the 6-week safety threshold.
The Hypervolemia Trap
Unlike pure testosterone, Dbol converts into **17α-methylestradiol**. This isomer is resistant to conventional Aromatase Inhibitors and causes aggressive intracellular water retention. The resulting **Hypertension** (High Blood Pressure) is often the silent catalyst for Left Ventricular Hypertrophy during high-dose cycles.
☢
Mandatory Forensic
Safety Notice
// HEPATIC INTEGRITYPersistent use of 17-alpha-alkylated steroids is correlated with malignant liver tumors and hepatic failure. Cycles must be limited to 4-6 weeks maximum.
// LEGAL REPERCUSSIONSMethandrostenolone is a Schedule III controlled substance. Possession without a clinical diagnosis of muscle wasting is a felony offense in multiple jurisdictions.
// UGL CONTAMINATIONUnderground Dbol is frequently cut with Methyltestosterone or contaminated with heavy metals from low-grade synthesis environments.
STOP
SYSTEM LOCKOUTAbsolute Contraindications for Methandrostenolone
Non-Alcoholic Fatty Liver (NAFLD)FATAL RISK
Dbol induces intrahepatic cholestasis. Adding it to a pre-existing fatty liver environment triggers acute liver enzyme spikes (>300 ALT) and potential organ failure.
Stage 1/2 HypertensionVASCULAR CRITICAL
If baseline BP is >130/80, Dbol is a 'Stroke-in-a-bottle'. The rapid sodium retention will push systolic levels into the hypertensive crisis zone (180+).
Gynaecomastia SensitivityENDOCRINE ALERT
Dbol aromatizes into 17-Methylestradiol, which has a higher affinity for breast tissue receptors than standard estrogen. Rapid-onset 'Gyno' is highly likely.
Kidney Filtration IssuesRENAL LOCK
Elevated creatinine or low eGFR (<60) makes Dbol use impossible. The resulting fluid shift places unmanageable hydrostatic pressure on the glomeruli.
🦴
Permanent Stunting: The Growth Plate LockdownDianabol’s high aromatization rate causes an immediate spike in circulating estrogen. For users under 25, this triggers the **irreversible ossification** (closing) of the epiphyseal plates. Once closed, height growth is biologically impossible, regardless of GH levels or nutrition.